スタットパール

/ /

日本語AIでPubMedを検索

PubMedの提供する医学論文データベースを日本語で検索できます。AI(Deep Learning)を活用した機械翻訳エンジンにより、精度高く日本語へ翻訳された論文をご参照いただけます。

スタットパール

StatPearls

James Knight
Lawrence C. Decker

PMID: 32644561

抄録

姿勢の変化は、体幹と四肢のステレオタイプの動きを伴い、典型的には重大な脳や脊髄の損傷を示している[1]。[1]ノーベル賞受賞者であるチャールズ・シェリントンは、生きたサルとネコの脳を横断した後、1898年に初めて減脳性姿勢を記述した[2]。2] decerebrate posturingの同義語には、異常伸展、decerebrate硬直、伸筋姿勢、またはdecerebrate反応が含まれる。いくつかの解剖学的領域の脳病変が両方の姿勢を引き起こす可能性があるが、通常はある程度の脳幹の損傷を伴うものである[1]。ほとんどの文献では、このレベルは中脳の毛根間レベルの赤核と考えられている[2][3]。

Decorticate and decerebrate posturing are both considered pathological posturing responses to usually noxious stimuli from an external or internal source. Both involve stereotypical movements of the trunk and extremities and are typically indicative of significant brain or spinal injury.[1] The Nobel Laurette Charles Sherrington first described decerebrate posturing in 1898 after transecting the brainstems of live monkeys and cats.[2] Synonymous terms for decorticate posturing include abnormal flexion, decorticate rigidity, flexor posturing, or decorticate response. Synonymous terms for decerebrate posturing include abnormal extension, decerebrate rigidity, extensor posturing, or decerebrate response.[2] There is a criticism within the literature of the use of the terms decorticate and decerebrate posturing in clinical contexts due to their association with discrete anatomical locations that, in reality, may not be so prescriptive. Brain lesions of several anatomical regions may cause both postures, though they do usually involve some degree of brainstem injury.[1] It is, however, accepted that decorticate typically requires an injury more rostral than decerebrate posturing. In most literature, this level is considered the red nucleus at the intercollicular level of the midbrain.[2][3]